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CVB3 is included in the genus Enterovirus within the family Picornaviridae and is a major causative agent of cardiac muscle infection, but the mechanism is still unclear (61)

Notably, patients in this trial experienced significantly less worsening of UPDRS scores compared to historical controlled studies

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Missense variants in FIC-related genes are associated with both PFIC and milder disease forms like BRIC, depending on the specific amino acid alteration and the patients genotypic status (homozygous, compound heterozygous, or heterozygous) [19]